Postural orthostatic tachycardia syndrome (POTS).The shock resets the heart and returns it to its normal rhythm. Treatment is an electric shock to the heart, using a machine called a defibrillator. Without treatment, ventricular fibrillation may be fatal. This causes a series of rapid – but ineffective – contractions of the ventricles. Like atrial fibrillation, the electrical signal that normally triggers a heartbeat splits and goes off on random paths around the ventricles instead of following its normal route. Incorrectly timed electrical signals or signals that do not follow normal pathways, may set off ventricular fibrillation. Other treatments include an implantable defibrillator, catheter ablation, non-surgical procedures to destroy malfunctioning cells, or surgery to remove damaged heart tissue. Ventricular tachycardia can be treated with medications. If tachycardia becomes so severe that the ventricles can't pump effectively, it may be life threatening. The ventricles are responsible for pumping blood to the rest of the body. Ventricular tachycardia occurs when the ventricles (the lower chambers of the heart) beat too fast. Other symptoms include palpitations, shortness of breath, fainting and possibly angina. Some people experience rapid heartbeats (paroxysmal supraventricular tachycardia), with heart rates rising up to 240 beats per minute. Many have no symptoms and have no episodes of tachycardia. Most people with WPW syndrome lead normal lives. The abnormality is present at birth (congenital), but WPW is usually diagnosed in adolescence or early adulthood. If you have WPW syndrome, an extra, abnormal electrical pathway in your heart leads to tachycardia. It may be distressing, but it is rarely life-threatening. PSVT often starts when you are young, but it may also start later in life. PSVT usually occurs in people who are born with an extra electrical circuit or pathway between the atria and the ventricles. This type of tachycardia produces heart rates between 140 and 250 beats per minute.
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